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These are datasets, gene expressions from Affymetrix microarrays, previously uploaded by a logged user.

Click on Details to see aditional information about the selected dataset.
From there you can explore the Gene Expression among the different data preprocessing.

Human tissue     Details 54675 genes x 13 experiments
Description:
Different human tissues like blood, skin and muscles.
Species:
Homo Sapiens
Input data type:
Microarray, HG-U133 Plus
Human muscle     Details 54675 genes x 8 experiments
Description:
Different human muscle tissue: Vastus lateralis, quadriceps, deltodis and vascular smooth muscle.
Species:
Homo Sapiens
Input data type:
Microarray, HG-U133 Plus
Arabidopsis Selenate     Details 22810 genes x 8 experiments
Description:
Gene expression in roots and shoots of plants grown on selenate. http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE9311
Species:
arabidopsis thaliana
Input data type:
Microarray, ATH1-12501
Translating Dosage Compensation to Trisomy 21     Details 49395 genes x 27 experiments
Description:
Down syndrome is a common disorder with enormous medical and social costs, caused by trisomy for Chr21. We tested the concept that gene imbalance across an extra chromosome can be de facto corrected in DS patient stem cells by manipulating a single gene, XIST. Using zinc finger nucleases, we targeted a large, inducible XIST transgene into the Chr21 DYRK1A locus, in DS pluripotent stem cells. XIST RNA coats Chr21 and triggers stable heterochromatin modifications, chromosome-wide transcriptional silencing and DNA methylation to form a “Chr21 Barr Body.” This provides a model to study human chromosome inactivation and creates a system to investigate genomic expression changes and cellular pathologies of trisomy 21, free from genetic and epigenetic noise. In this study, we used microarrays to understand the genome-wide impacts of inducible XIST expression on Chr21 in trisomy 21 human iPS cell lines, and to evaluate the extent of Chr21 silencing trisomic samples versus a disomic male iPS cell line.
Species:
Homo Sapiens
Input data type:
Microarray, PrimeView
[Chr21]Translating Dosage Compensation to Trisomy 21     Details 610 genes x 27 experiments
Description:
Down syndrome is a common disorder with enormous medical and social costs, caused by trisomy for Chr21. We tested the concept that gene imbalance across an extra chromosome can be de facto corrected in DS patient stem cells by manipulating a single gene, XIST. Using zinc finger nucleases, we targeted a large, inducible XIST transgene into the Chr21 DYRK1A locus, in DS pluripotent stem cells. XIST RNA coats Chr21 and triggers stable heterochromatin modifications, chromosome-wide transcriptional silencing and DNA methylation to form a “Chr21 Barr Body.” This provides a model to study human chromosome inactivation and creates a system to investigate genomic expression changes and cellular pathologies of trisomy 21, free from genetic and epigenetic noise. In this study, we used microarrays to understand the genome-wide impacts of inducible XIST expression on Chr21 in trisomy 21 human iPS cell lines, and to evaluate the extent of Chr21 silencing trisomic samples versus a disomic male iPS cell line.
Species:
Homo Sapiens
Input data type:
Microarray, PrimeView
Random example     Details 100 genes x 10 experiments
Description:
Random matrix example with 3 clusters.
Species:
Input data type:
Microarray,
Breast_A     Details 1213 genes x 97 experiments
Description:
Microarray data from Broad Institute “Cancer Program Data Sets” which was produced by (van’t Veer et al., 2002) (http://www.broadinstitute.org/cgi-bin/cancer/datasets.cgi) Array S54 was removed because it is an outlier.
Species:
Homo Sapiens
Input data type:
Microarray, HG-U133 Plus